No structural fetal
anomalies. It appears that the frequency of congenital malformation after
infection is not increased as compared with the general population (1).
Hepatic fibrosis and
necrosis (either due to direct effect on hepatocytes or indirect damage
from hemosiderin deposition in hepatocytes (3).
Symmetric IUGR (2). The fetal growth
appears to catch up once the hydrops resolves (4).
Torok TJ, Wang Q-Y, Gary GW
Jr et.al. Prenatal diagnosis of intrauterine infection with parvovirus B19
by the polymerase chain reaction technique. Clin Infect Dis
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Bhal PS, DaviesNJ,
Westmoreland D et.al. Spontaneous resolution of non-immune hydrops fetalis
secondary to transplacental parvovirus B19 infection. Ultrasound Obstet
Gynecol 1996;7:55-57.
Kilham L, Margolis G.
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neonatal infections due to viruses. Prog Med Virol 1975;20:113.
Pryde PG, Nugent CE,
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